About Your Bones and Osteoporosis
Bone is a living organ that is in a constant cycle of renewal and removal. During the aging process, bone replacement (growth) slows down while bone removal (resorption) accelerates. This is particularly true for women during and after menopause.2
Osteoporosis is a progressive disease that occurs when new bone creation is unable to keep up with bone loss.3 This causes bones to become porous and brittle, increasing the risk of fractures. Typically asymptomatic or “silent,” the condition is commonly not detected until the occurrence of a fracture.
Osteoporosis can be caused by various factors. The most common cause is the lack of estrogen and testosterone hormones.4 Other factors include lack of dietary calcium and vitamin D, smoking, and certain medical conditions such as diabetes.2 Low bone mass affects about half of Americans over 50, with the majority of them being women.
Diagnosis is achieved primarily through the use of a Bone Mineral Density assessment, also known as a DEXA (or DXA) scan or a bone density scan.
Osteoporotic fractures typically occur in the hip, spine, or wrist. While not a direct cause of death, fractures in the hip and spine severely limit independence, and are associated with a mortality rate of 33% and 20% after one year for men and women, respectively.5
Conventional Treatment Options
First-course treatment options include lifestyle changes and various pharmaceutical treatments, depending on the cause of bone loss. Lifestyle changes usually include diet changes to increase calcium and vitamin D intake, as well as undertaking weight-bearing and resistance exercises such as walking or jogging and weightlifting. Medical therapies include bisphosphonates to help increase bone density and hormone replacement therapy to help slow down bone loss.6 Possible treatments are best discussed with your physician.
Where Vibration Therapy Fits In
Based on NASA technology developed to prevent bone loss for astronauts in zero gravity, vibration therapy devices simulate the mechanical loading of your bones during exercise, which stimulates bone growth. Vibration therapy has been shown in several clinical studies to effectively encourage bone growth and prevent bone loss in the the hips and spine.7,8
Because vibration therapy is drug-free, it has no known side effects and will not interact with any current prescriptions medications or other therapeutic approaches. This makes vibration therapy the perfect complementary treatment option in the fight against low bone density.
Clinically proven to decrease bone loss activity.
In a clinical trial conducted at the University of Nebraska Medical Center, OsteoBoost’s targeted vibration therapy decreased bone loss activity by an average of 14% in just one treatment. This is consistent with the effects of traditional treatment options, validating OsteoBoost as an effective drug-free alternative in the fight against age-related bone loss.
Clinical Trial Details
In collaboration with Dr. Laura Bilek at the University of Nebraska Medical Center, Bone Health Technologies conducted a clinical trial of OsteoBoost in 17 postmenopausal women. This trial evaluated the effect of the 30-minute treatment session on NTX, a biomarker of bone removal, a process that contributes to lower bone density with age.
Change in NTX levels via blood draws was noted in sessions during which the device was not turned on (control), and another during which each participant experienced a 30-minute OsteoBoost session. In all 17 women, the OsteoBoost targeted vibration therapy produced a decrease in NTX compared to the control session, with an average decrease of 14%. This is consistent with NTX changes provided by traditional treatment options such as drug therapy and exercise. This clinical trial validates OsteoBoost as an effective drug-free alternative for combating bone loss and improving bone health.
“OsteoBoost is easy to use and it actually feels good on my back. I keep it in my kitchen and now it’s just a part of my morning routine. It’s like giving my bones an extra 30-minute workout every day – what’s not to love?”
“OsteoBoost is super easy – easy to wear, quieter than I thought it would be, and has an added bonus of relaxing my low back muscles. I’ve been using it every day and like that it’s helping my bone health.”
“The beauty of the OsteoBoost wearable technology is that it is easy to add to a daily routine, there is no downside, and it offers vast potential to build better bones.”
Dr. Shane Mangrum, Physical Medicine & Rehabilitation
1. Martyn-St James M, Carroll S. (2008). Meta-analysis of walking for preservation of bone mineral density in postmenopausal women. Bone, 43(3);2008;521-531. doi: 10.1016/j.bone.2008.05.012
2. Osteoporosis. (2019, June 19). Retrieved February 24, 2020.
3. Learn what osteoporosis is and what it’s caused by. (n.d.). Retrieved February 25, 2020.
4. Demontiero O, Vidal C, Duque G. (2011). Aging and bone loss: new insights for the clinician. Therapeutic Advances in Musculoskeletal Disease, 4(2), 61–76. doi: 10.1177/1759720×11430858.
5. Lisk R, Yeong K. (2014). Reducing mortality from hip fractures: a systematic quality improvement programme. BMJ Quality Improvement Reports, 3(1). doi: 10.1136/bmjquality.u205006.w2103.
6. Eastell R, Rosen CJ, Black DM, Cheung AM, Murad MH, Shoback D. (2019). Pharmacological management of osteoporosis in postmenopausal women: an endocrine society* clinical practice guideline. The Journal of Clinical Endocrinology & Metabolism, 104(5), 1595–1622. doi: 10.1210/jc.2019-00221.
7. Slatkovska L, Alibhai SMH, Beyene J, Cheung AM. (2010). Effect of whole-body vibration on BMD: a systematic review and meta-analysis. Osteoporosis International, 21(12), 1969–1980. doi: 10.1007/s00198-010-1228-z.
8. Lebrun C. (2012). Effects of whole-body vibration training on different devices on bone mineral density. Yearbook of Sports Medicine, 2012, 419–420. doi: 10.1016/j.yspm.2012.03.042.
9. Gómez-Cabello A, Ara I, González-Agüero A, Casajús J, Vicente-Rodríguez G. (2012). Effects of training on bone mass in older adults. Sports Medicine, 42(4), 301–325. doi: 10.2165/11597670-000000000-00000.